BEGINNER ROUTE · MOVE ANYWHERE WITHOUT LOSING YOUR PLACE

Choose a lesson.

CHAPTER 01 OF 27See the whole scanner

Start with the patient, table, nested scanner hardware, and the order in which an MRI measurement happens.

Theme 03 · Signal, contrast and motion

Run a visual exam from coil setup and localizers through slice prescription, sequence contrast, modeled k-space, and reconstruction—then inspect the physics underneath.

Choose receive hardware, acquire three-plane localizers, prescribe and reconstruct synthetic T1/PD/T2/FLAIR/DWI/GRE anatomy, then explore echo formation, steady states, perfusion, flow, angiography, and diffusion.

View the complete lab

03 / SIGNAL, CONTRAST & MOTION

Wait for recovery.
Encode displacement.

Start with the integrated virtual exam: choose receive hardware, acquire localizers, prescribe a slice, tune T₁/PD/T₂/FLAIR/DWI/GRE controls, and reconstruct modeled k-space. TE, TR, flip angle, and RF history shape the available signal. BOLD fMRI turns a delayed vascular oxygenation consequence into T₂*-weighted time-series change; ASL magnetically labels arterial blood to estimate perfusion; TOF turns replacement of saturated blood into bright-vessel magnitude; phase contrast turns coherent motion into signed velocity; diffusion sensitization probes microscopic displacement. These are distinct mechanisms—not interchangeable maps.

01 · VIRTUAL EXAM + RELAXATION HISTORYCoil setup, slice geometry, and pulse timing meet in one reconstruction.

Localize and prescribe first; then use the same quantitative sequence controls to acquire T₁, proton-density, T₂, FLAIR, DWI, or GRE contrast.

02 · FUNCTIONAL HEMODYNAMICSNeural input changes flow, volume, and deoxyhemoglobin before GRE signal.

BOLD fMRI is delayed and indirect: a vascular state changes susceptibility-related T₂* weighting, then EPI samples volumes at TR.

03 · LABELED-BLOOD PERFUSIONPrepared arterial spins travel, decay, arrive, and subtract.

ASL couples label duration, ATT, PLD, T₁ survival, control−label difference, and model-based CBF.

04 · BRIGHT-BLOOD INFLOWFresh replacement outruns repeated-RF saturation.

TOF couples slab-normal transport to spoiled-GRE history, source magnitude, directional preparation, and MIP.

05 · COHERENT FLOWOpposite first moments turn velocity into signed phase.

Phase contrast subtracts two complex measurements, decodes one component, and exposes VENC aliasing and area integration.

06 · DIFFUSION SENSITIZATIONMatched gradients cancel for stationary spins.

Microscopic displacement between lobes leaves a phase distribution; a wider distribution reduces the coherent echo.

07 · DIRECTIONAL CONTRASTOne gradient direction probes one tensor projection.

Repeat directions to estimate anisotropy; one direction and one b-value are not a tensor fit or tract map.

3D ECHO FORMATION · ONE VOXEL IN THE ROTATING FRAME

A 180° pulse can reverse static phase spread.
It cannot rewind true T₂ loss.

Follow the same transverse ensemble through excitation, dephasing, a refocusing action, rephasing, the echo, and renewed dephasing. Switch mechanisms without changing the tissue or field spread.

REVERSIBLE ↔ IRREVERSIBLE DEPHASINGSTATIC Δf + INTRINSIC T₂ · ANALYTIC LORENTZIAN TEACHING MODEL
01 / 06 · EXCITATION COMPLETESTART WITH COHERENT TRANSVERSE PHASE

A 90° excitation is represented as complete. Every visible spin packet begins along +X′ in a frame rotating near the carrier; the fast MHz carrier itself is removed.

3D

The rotating-frame echo model needs WebGL. The synchronized timeline, equations, metrics, and controls remain available.

Measured time stays 2DSPIN ECHO · 180° AT 40.0 ms · ECHO AT 80 ms
Echo-formation event timeline Spin echo with excitation at zero, a 180 degree pulse at half the echo time, and a signal maximum at the chosen echo time. ACTION PHASE SIGNAL 90° 180° RF TE · 80 ms

The pulse or gradient command is a cause. The phase fan converges later. Vertical screen position is a teaching state—not another scanner coordinate.

0% · 0.0 ms exciterefocus at 50% · echo at 100%after

This teaching scrubber changes the instant being inspected. It does not change the prescribed TE, tissue, or field spread.

80 ms 20 ms180° / reversal at TE ÷ 2160 ms

Longer TE gives both irreversible T₂ relaxation and unrefocused static offsets more time to reduce the gradient echo. The ideal spin echo removes the static-offset term only at its echo center.

4.0 Hz 0 HzLorentzian reversible offset distribution20 Hz

A wider static frequency distribution fans phase faster. The 180° pulse reverses its ordering; a gradient-polarity reversal does not.

IDEAL SPIN ECHO AT TE

SSE(TE) / S₀ = e−TE/T₂

The 180° pulse removes the static-offset phase factor at TE; it does not restore the T₂ envelope.
DECLARED LORENTZIAN FIELD-SPREAD MODEL

1/T₂* = 1/T₂ + 1/T₂′  ·  T₂′ = 1/(πΔfFWHM)

1/T₂* = 1/80 + 1/79.6 ms → T₂* = 39.9 ms
CURRENT TIME / ACTION0.0 ms · excitation completeall visible packet phases begin aligned
INTRINSIC COHERENCE ENVELOPE1.000T₂ survival at the inspected time
REVERSIBLE STATIC COHERENCE1.000static Δf phase factor in spin-echo mode
MODELED CURRENT |S| / S₀1.000analytic envelope; finite visible arrows are a sampling cue
IDEAL SPIN ECHO AT TE0.368T₂ loss remains after static refocusing
GRADIENT ECHO AT SAME TE0.135T₂* includes unrefocused static spread
DERIVED T₂′ / T₂*79.6 / 39.9 msreversible-only / combined apparent constant
SIGNAL RECOVERED BY IDEAL 180°+0.233SE − GRE at the same TE; not recovered T₂ loss
SPIN ECHO · T₂36.8%
GRADIENT ECHO · T₂*13.5%

WHY THE TWO ECHOES DIFFER ·At TE 80 ms, intrinsic T₂ leaves 36.8%. A 4.0 Hz static spread reduces the gradient echo to 13.5%, while an ideal 180° pulse removes that static factor at the spin-echo center.

01 · REVERSIBLE

Static frequency offsets keep their sign.

Fast packets move ahead and slow packets fall behind. An ideal 180° pulse swaps that phase ordering, so continued evolution brings them together at TE.

02 · IRREVERSIBLE

True T₂ coherence keeps decaying.

The visible packet arrows shorten as the modeled coherent contribution survives. The refocusing pulse changes future phase evolution; it does not add lost coherence or reset time.

03 · MECHANISM MATTERS

Opposite gradient area is not a 180° pulse.

A gradient echo cancels the deliberate gradient moment at TE but leaves static B₀ offsets accumulating, so its ideal envelope follows T₂*.

TE / TR RELAXATION CLOCKILLUSTRATIVE 3 T TISSUE MODEL · 128 PHASE ENCODES
VIRTUAL MRI EXAM · SYNTHETIC TEACHING WORKSTATION

Choose receive hardware, localize, prescribe a slice, then acquire it.

Review the 3D RF + Gz slice-selection microscope →

LIVE SEQUENCE · T₁ SPIN ECHOTR 500 · TE 15 ms

Start with a study · then change one thingILLUSTRATIVE TEACHING STATES · NOT PROTOCOLS

HEAD TEACHING STARTBrain, ventricles, and posterior fossa. Acquire the synthetic localizers, prescribe, then run the virtual scan.

Interactive 3D coil and slice setup
COIL SETUPHEAD / NECK
3D LAYERS
Receive hardware · representative named examplesSELECT ONE SETUP
HEAD / NECK 2020 receive channelsopen-face head helmet with neck extension
TRANSMIT
built-in body transmit
RECEIVE
circumferential head and upper-neck receive array
CENTER SENSITIVITY
1.00× relative
UNIFORMITY PROXY
0%
MEAN RECEIVE PROXY
0.00× effective
PARALLEL IMAGING
not simulated

Receive proxy = mean analytic sensitivity over one central axial elliptical mask. Nominal channel count is a hardware label, not a multiplier. A posterior partner changes the modeled sensitivity term. This does not predict clinical SNR or usable acceleration.

THREE-PLANE LOCALIZER / SCOUTNOT YET ACQUIRED
SAGITTALA/P × H/F
CORONALR/L × H/F
AXIALR/L × A/P

Acquire the survey first. Localizers reveal internal synthetic anatomy relative to scanner coordinates; the external landmark alone does not prescribe a slice.

ONE 3D MODEL, THREE REPARAMETERISATIONSAll three surveys and every prescribed slice sample the same code-native 3D tissue field in patient millimetres: R/L positive toward L, A/P positive toward P, H/F positive toward F, measured from each region's modelled isocentre. Axial reads (R/L, A/P) at a fixed H/F, sagittal reads (A/P, H/F) at a fixed R/L, coronal reads (R/L, H/F) at a fixed A/P. R→L runs left to right; axial A→P and sagittal/coronal H→F run top to bottom. Positive slice position points toward L, P, or F for sagittal, coronal, or axial prescriptions, and its limit is that region's own half-extent along the selected normal. Each survey is a soft-maximum (power-mean, p = 3) projection of tissue proton density ρ through a 15 mm slab. Every displayed scout voxel uses 2 × 2 in-plane midpoint samples and seven through-slab samples—28 samples total. It is a declared projection, not a simulated pulse sequence, so it carries no TR, TE, TI, flip angle, relaxation, diffusion, coil weighting, or noise. Survey brightness uses a declared display window (ρ 0.56–1.02, γ 0.90); the prescription overlay is drawn in true millimetres at each survey's own scale.

Slice prescriptionWAITING FOR LOCALIZER
−20 mm −110H/F axis+110 mm
−45°relative teaching rotation+45°
5.0 mm 1 mmthrough-plane averaging12 mm
12 1stack coverage32
1.0 mm 0not tissue removed8 mm
220 mm 100read × phase coverage520 mm
PIXEL1.72 × 1.72 mm
VOXEL14.8 mm³
RELATIVE SNR1.00× preset
THROUGH-PLANE DETAIL4.0 mm samples
STACK COVERAGE71 mm
SCAN-TIME PROXY1:04
ONE-CHANGE EXPERIMENTScompare with this study's starting state

AT THE TEACHING STARTChange slice thickness, FOV, matrix, or NEX. This readout separates resolution, SNR, coverage, and time so “brighter” is not mistaken for “better.”

CONTRAST FOR THE NEXT ACQUISITIONThese buttons drive the same anatomy and quantitative controls below.Fine-tune TR / TE / TI / b / ADC / flip ↓
Physiologic motion + triggeringLINE-WISE FOURIER PHASE MODEL
70 /min 35cycles per minute180
0 mm 0phase-encode direction30 mm
20% 5%of each physiology cycle80%
0% 0%timing jitter + rejected windows40%
STILL PATIENTNO PHYSIOLOGIC DRIVER
CYCLE
LINES / WINDOW
all lines
RESIDUAL MOTION
0.0 mm
WALL-CLOCK PROXY
1:04

NO PERIODIC MOTIONThe k-space rows remain mutually consistent. Choose Cardiac or Respiratory, then compare gating on and off in the reconstructed image.

VIRTUAL ACQUISITION · FOURIER RECONSTRUCTION FROM ACQUIRED LINESREADY AFTER LOCALIZER + PRESCRIPTIONEach acquired phase-encode line adds modeled complex k-space values for the selected center slice; the evolving image is recalculated from only those lines, with unacquired lines set to zero.
ACQUIRED k-SPACE0 / 128 kᵧ lines
SELECTED CENTER SLICE · PARTIAL RECONSTRUCTIONNO ACQUISITION YET

WHAT IS QUANTITATIVETR, TE, TI, flip angle, b/ADC signal equations; FOV/matrix pixel size; voxel volume; slice-stack coverage; one-line-per-TR base time; SNR scaling with voxel volume and √NEX while other factors are held fixed; the exact Fourier translation phase applied to each modeled kᵧ line; the Fourier transform of the displayed center-slice synthetic source; and the plane geometry itself — every survey, overlay, embedded 3D slice, and acquired image is the same 3D field sampled through one millimetre coordinate map.

WHAT IS ILLUSTRATIVEThe ten study buttons are starting states, not protocols. Analytic coil sensitivity, deterministic complex k-space noise, the cardiac/respiratory waveform, trigger acceptance and wall-clock proxy, whole-slice rigid displacement, procedural anatomy, derived display shells, localizer projection/window, partial-volume quadrature, preview resolution, per-region scene scale, nominal channel labels, and compressed playback are teaching models. Slice thickness can improve this model's SNR and increase through-plane averaging; fixed display windowing means it does not guarantee a brighter image.

WHAT IS OMITTEDThe other prescribed slices and repeated NEX lines are not separately reconstructed. The anatomy is hand-declared—not segmented, measured, atlas-derived, registered, diagnostic, or population-normal. Motion is one rigid phase-axis translation, not organ deformation, flow, temporal cine reconstruction, navigator design, arrhythmia classification, prospective controller behavior, or a patient-specific quiet window. Also omitted: measured B₀/B₁/coil maps, channel noise covariance, bandwidth changes, parallel reconstruction, safety screening/limits, calibration, and vendor protocol logic. Nothing here recommends a clinical protocol.

Shared synthetic axial anatomy · live signalT₁ SPIN ECHO · TR 500 / TE 15 ms
ANATOMY PROBE · SAME LOCATION THROUGH EVERY SEQUENCE RESTRICTED-DIFFUSION CORE ρ 0.92 · T₁ 1500 · T₂ 120 ms · ADC 0.45 × 10⁻³ mm²/s current relative signal 0.000
fatgraywhiteCSFT₂ lesionrestricted core
Recovery before RF / decay before echoT₁ RECOVERY + T₂ DECAY
IDEAL SPIN-ECHO SIGNAL

S = ρH(1 − e−TR/T₁)e−TE/T₂

The 180° pulse refocuses static dephasing, so ideal echo amplitude follows T₂ rather than T₂*.
500 ms 200 msrecovery + scan time6000 ms
15 ms 5 mstransverse decay200 ms
90° fixed spin echo fixes 90°60°
WHITE MATTERρ 0.70 · T₁ 850 · T₂ 80 · T₂* 55 ms
CSF / FLUIDρ 1.00 · T₁ 4000 · T₂ 2000 · T₂* 400 ms
One repetition · event spacing compressed90° → 180° → ECHO → NEXT 90°
RFGdSIG 90° 180° TE 15 ms TR 500 ms
SHORT TR · SHORT TE

Short TR samples tissues before full longitudinal recovery, strengthening T₁ differences. Short TE limits T₂ decay, so the current spin echo is predominantly T₁ weighted.

white matter signal0.280
CSF / fluid signal0.114
pairwise contrast42.2%
dominant weightingT₁ weighted
2D scan-time proxy1:04 · 128 lines
echo fraction TE / TR3.0%
WHITE0.000
GRAY0.000
CSF0.000
FAT0.000
T₂ LESION0.000
RESTRICTED CORE0.000

MODEL BOUNDARY · SYNTHETIC ANATOMY, NOT A PATIENT OR PROTOCOL RECOMMENDATION. Every pixel belongs to one ideal nonexchanging tissue compartment with illustrative values near 3 T. Spin echo uses ρ(1−e−TR/T₁)e−TE/T₂; FLAIR adds one ideal inversion-prepared repeated cycle; DWI multiplies the same spin-echo baseline by e−b·ADC; GRE uses the spoiled steady state and T₂*. The grayscale uses one fixed relative-signal display mapping, so numeric comparisons remain available without per-preset auto-windowing. Real anatomy and contrast also depend on field strength, sequence family, echo trains and k-space ordering, inversion and fat-suppression profiles, diffusion direction, T₂ shine-through, perfusion, flow, exchange, coils, noise, artifacts, reconstruction, pathology, safety limits, and vendor timing constraints.

ADVANCED 3D MODEL · T₁ SATURATION ACROSS REPETITIONS

One RF pulse is simple.
The history creates the steady state.

Follow one ideally spoiled gradient-echo experiment pulse by pulse. The vector shortens inside a reference sphere, the history plot records the exact recurrence, and the Ernst protractor shows which flip angle maximizes ideal steady-state signal for the selected tissue and TR.

ROTATING FRAME · Z ∥ B₀ · NORMALIZED M / M₀ Before RF · repetition 1

The experiment begins at equilibrium: Mz⁻ = 1 before the first RF pulse. The translucent sphere is only the M₀ reference boundary.

The quantitative history plot remains available if 3D rendering is unavailable.

DRAG TO ORBIT · + / − TO ZOOM · TAP AN OBJECT TO IDENTIFY IT

3D LADDER · ONE SHARED SCALE Mz⁻ pre-RF Mz⁺ post-RF Mxy⁺ immediate S(TE) readout Each column is one repetition. Ideal spoiling sets coherent Mxy to zero before the next RF pulse.
Quantitative repetition history 2D PLOT IS THE NUMERIC SOURCE OF TRUTH
tissue A · Mz⁻ before RF tissue A · Mxy just after RF tissue B · Mz⁻ before RF tissue B · Mxy just after RF

Important: these curves show magnetization state, not TE-weighted signal. TE scales the echo readout after each pulse; it does not change convergence toward the spoiled-GRE steady state.

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12° near WM Ernst angle60°
20 ms 5 msT₁ recovery between tips100 ms
5 ms 1 mssignal only · not convergence19 ms
IDEALLY SPOILED GRE · DISCRETE LONGITUDINAL RECURRENCE

Mn+1 = 1 − (1 − Mn cosα)E₁

E₁ = e−TR/T₁ · fixed point Mss = (1 − E₁)/(1 − E₁ cosα) · αE = cos−1(E₁)
WHITE MATTER · CURRENT Mz⁻1.000pulse 1 · 100% of initial error remains
steady Mz⁻0.521within 5% by pulse 67 · beyond rail
Ernst angle · tissue A12.4°TR 20 ms · T₁ 850 ms
selected α − Ernst−0.4°near the ideal signal maximum
Mxy immediately after RF0.208state before T₂* weighting
echo signal at TE0.133ρ × Mxy × e⁻ᵀᴱ/ᵀ²*
WHITE MATTER · SOLIDT₁ 850 ms · αE 12.4°Sss 0.069
GRAY MATTER · DASHEDT₁ 1350 ms · αE 9.8°Sss 0.068
01 · BEFORE RFHistory sets available Mz⁻.

The first repetition starts at equilibrium. Later pulses begin from the recovery left by every earlier pulse.

02 · RF TIPα partitions the vector.

Instantaneous ideal RF creates Mxy = Mz⁻ sinα and leaves Mz⁺ = Mz⁻ cosα.

03 · READ + RESETTE weights; the ideal model resets coherence.

T₂* reduces the measured echo. The recurrence then applies coherent Mxy → 0 before the next pulse without claiming a gradient- or RF-spoiling mechanism.

04 · T₁ RECOVERYThe recurrence contracts.

During TR, Mz returns toward M₀. Repeating the same map approaches one fixed point at rate E₁ cosα.

MODEL BOUNDARY · IDEALLY RF-SPOILED GRE, NOT A FULL BLOCH OR bSSFP SIMULATOR

One normalized isochromat per tissue is shown in the rotating frame, with instantaneous nonselective RF, perfect loss of coherent transverse magnetization between repetitions, monoexponential T₁ recovery, and magnitude-only T₂* echo weighting. T₁ recovery during the short TE is folded into the repetition map. The model excludes coherent/balanced SSFP, RF phase cycling details, B₁ error, slice profile, inflow and flow, magnetization transfer, diffusion, exchange, multiple compartments, off-resonance banding, imaging gradients, noise, and reconstruction. Z/B₀ is the spin-frame reference here, not a patient-orientation claim.

ADVANCED 3D MODEL · INVERSION RECOVERY FAMILY

Invert first.
Wait for one tissue to disappear.

An inversion preparation sends longitudinal magnetization below zero. Tissues recover at different T₁ rates, so a readout placed at the right inversion time can null fat, null fluid, or preserve the sign that ordinary magnitude display folds away.

INVERTMove Mz toward −M₀.

Efficiency controls the effective longitudinal inversion; it is not drawn as a fictitious hard-pulse angle.

RECOVEREach tissue crosses zero at its own TI.

The finite-TR fixed cycle shifts that crossing from the familiar long-TR T₁ ln 2 result.

READSigned and magnitude displays are different.

A phase-sensitive display preserves negative-versus-positive recovery; magnitude folds both sides above zero.

LONGITUDINAL FRAME · NORMALIZED M / M₀ · MATERIALS, NOT ANATOMY Compare tissue-specific zero crossings

The five declared materials rise through the shared zero plane at different times; fat is at its finite-TR crossing near TI 243 ms.

05 / 05 Mz +0.001 fat · TI 243 ms

The signed TI-response plot remains available if 3D rendering is unavailable.

DRAG TO ORBIT · + / − TO ZOOM · TAP AN OBJECT TO IDENTIFY IT

Signed longitudinal state + displayed echo versus TI EXACT NUMERIC RESPONSE · CURRENT TR AND READOUT
white matter gray matter CSF / fluid fat custom lesion target / comparator echo

Upper plot: signed Mz immediately before the readout. Lower plot: the chosen signed or magnitude echo after flip-angle, proton-density, and TE weighting.

243 ms inversionnear fat nullreadout
4000 ms 800sets next preparation history12000 ms
60 ms 0T₂ spin-echo survival180 ms
1.00 0.50 · saturationcos θeff = 1 − 2η1.00 · ideal
90° tip Mz into measurable Mxy90°
1200 ms 300same declared PD / T₂ / T₂*4500 ms
EXACT SINGLE-COMPARTMENT PRE-READOUT STATE · REPEATED REGIME

Mz(TI) = 1 − cinvETR − (1 − cinv)ETI1 − cinvcos α ETR

cinv = 1 − 2η · ETI = e−TI/T₁ · S = ρ Mz(TI) sin α e−TE/T₂(or T₂*)
FAT · MAGNITUDE ECHO0.000spin echo / T₂ · TE 60 ms
signed Mz at TI+0.001pre-readout · above zero
exact target null243 msfinite-TR fixed cycle · long-TR 243 ms
distance from null0 mstarget is nulled
target − comparator−0.163WM +0.163
cycle closure< 10⁻¹²exact repeated fixed point
WHITE+0.163
GRAY+0.292
CSF+0.499
FAT0.000
LESION+0.319
01 · PREPAREInversion creates signed longitudinal contrast.

η = 1 maps +Mz to −Mz. Lower η reduces that effective longitudinal displacement and changes the null.

02 · SEPARATEShort-T₁ tissues recover first.

Fat crosses zero before white matter, gray matter, and CSF in this declared 3 T teaching table.

03 · SAMPLETI chooses the signed state; TE weights the echo.

The readout flip converts Mz into Mxy. Spin echo uses T₂; gradient echo uses T₂* in this model.

04 · DISPLAYMagnitude can hide which side of zero produced signal.

Signed mode retains polarity; magnitude mode applies an absolute value after the same physical signal calculation.

MODEL BOUNDARY · IDEAL SINGLE-COMPARTMENT INVERSION RECOVERY, NOT A PROTOCOL DESIGNER

Every material is one normalized, nonexchanging compartment with monoexponential T₁, T₂, and T₂* constants. Inversion and readout are instantaneous and spatially uniform. η is an effective longitudinal action, not a literal pulse angle or a model of adiabatic-pulse dynamics. The repeated mode contains one readout per TR with exact fixed-cycle history; fully recovered mode forces Mpre = M₀. The colored lanes and tiles are declared material samples, not anatomy or a diagnostic image. “STIR” and “FLAIR” presets isolate their nulling mechanism; they are not scanner protocols. “Signed / PSIR-like” preserves ideal signal polarity but does not model a reference acquisition, phase errors, or PSIR reconstruction. Excluded effects include slice profile, B₁ variation, finite pulse duration, magnetization transfer, exchange, multi-component fat, CSF flow, FSE echo trains, stimulated echoes, k-space ordering, coils, noise, motion, SAR, parallel imaging, and vendor timing constraints.

ADVANCED 3D MODEL · FSE / TSE / RARE · EXTENDED PHASE GRAPH

One excitation.
Many echoes, many pathways.

A fast spin-echo train is not one spin echo copied many times. Every refocusing pulse mixes transverse and longitudinal configuration states. Those coherent and stimulated-echo pathways determine each echo; assigning different echoes to phase-encode lines then changes contrast, point spread, and scan time.

CREATE + DEPHASEA 90° pulse creates F₀; gradients move coherence through configuration order.

EPG order k records accumulated gradient phase area. It is dimensionless pathway bookkeeping—not an imaging ky coordinate.

MIX + RECALLEach refocusing pulse redistributes F⁺, F⁻, and Z states.

Sub-180° pulses store part of the history longitudinally and recall it later as stimulated echoes.

ENCODE + RECONSTRUCTEcho index is mapped to a separate imaging ky line.

The resulting complex echo weighting is an MTF; its inverse transform is the phase-direction PSF.

TWO DIFFERENT SPACES ARE SHOWN—NEVER IDENTIFY THEM

EPG CONFIGURATION ORDER k tracks dephasing pathways inside one modeled compartment. IMAGING ky is a commanded spatial-frequency address acquired on one echo. The dashed bridge is a data-flow link, not an equality.

HARD-PULSE EPG · SIGNED COMPLEX STATES · SEPARATE GROUPED 1D ky ORDER Echo 8 · coherent pathways meet at F₀

The receiver samples only F₀ at 80 ms. Other transverse and longitudinal orders preserve hidden history for later echoes.

The exact echo-train, MTF, and PSF plots remain available if 3D rendering is unavailable.

DRAG TO ORBIT · + / − TO ZOOM · TAP AN OBJECT TO IDENTIFY IT

Echo train + refocusing schedule NORMALIZED |F₀| BEFORE ρ · DIRECT T₂ PATH IS A REFERENCE, NOT A CEILING
normalized pathway |F₀| · before proton density ρ normalized direct T₂ path · before ρ commanded refocusing angle
Declared echo-to-ky mapping → complex MTF → PSF SEPARATE IMAGING SPACE · 128 PHASE LINES
echo-index bands normalized |W(ky)| |inverse DFT W| source line weighted magnitude
16 echoes 48 shots for 128 ky lines32
10 ms 5 mstrain duration 160 ms20 ms
180° 50°constant hard pulses180°
8 / 16 first echoTE 80 mslast echo
8 early effective TEeffective TE 80 mslate effective TE
3000 ms 1000does not alter this single-train signal6000 ms
REFOCUSING PHASE
FLIP SCHEDULE
GROUPED 1D PHASE-ENCODE VIEW ORDER
HARD-PULSE EPG STATE TRANSITION + DECLARED IMAGING WEIGHT

Ωkafter RF = T(α,φ)Ωkbefore RF · Fk+ → Fk+1+

Sn = ρF0(n·ESP) · W(ky) = Se(ky)/maxe|Se| · PSF = DFT−1{W}
SELECTED ρ-SCALED COMPLEX ECHO |S|0.368phase −90.0° · echo 8
ρ-scaled direct T₂-path reference0.368EPG − direct +0.000
effective echo time80 msky zero |S| 0.368
longitudinal pathway energy0.0001 displayed configuration
PSF RMS width3.55 px90% energy width 3 px
largest PSF sidelobe30.3%|W| 0.153–1.000
scan time · 128 ky lines24.0 s8 shots · 6.25% of single-echo
numerical closure< 10⁻¹²Z symmetry + zero discarded state
01 · SHIFTGradient area advances transverse configuration order.

Relaxation scales F states by E₂ and Z states by E₁; recovery adds only to equilibrium Z₀.

02 · MIXRefocusing RF couples three complex coefficients at each order.

A 180° CPMG pulse swaps transverse pathways. Lower angles also create Z storage and later stimulated echoes.

03 · SAMPLEAn echo occurs when a pathway returns to F₀.

The receiver sees the signed complex sum at order zero, not the hidden higher-order states individually.

04 · MAPDifferent echo times weight different ky bands.

Center ky largely sets effective contrast; variation over ky determines the PSF, blur, ringing, and possible phase modulation.

MODEL BOUNDARY · IDEAL SINGLE-COMPARTMENT HARD-PULSE EPG, NOT A VENDOR SEQUENCE OR SAFETY CALCULATOR

The EPG engine is on-resonance, nonselective, single-compartment, and monoexponential. RF pulses are instantaneous and spatially uniform; every half-echo interval receives one identical positive unit gradient-order shift. CPMG and CP specify ideal RF-axis relationships. The “declared ramp” is a transparent exponential 180°-to-floor teaching schedule—not a vendor optimizer, prescribed-signal algorithm, or claim of an executable protocol. The imaging panel separately maps the solved echo train into declared grouped one-dimensional phase-encode bands across 128 lines. It is not the EPG configuration axis, a universal view order, or a two-dimensional anatomy reconstruction. TR is used only for scan-time accounting because every displayed train begins at equilibrium; inter-train saturation is excluded. Also excluded are slice profile and selective-pulse crushers, B₁/B₀ distributions, finite RF duration, chemical shift, multiple compartments, exchange, magnetization transfer, diffusion, flow, motion, noise, coils, parallel imaging, partial Fourier, echo sharing, ramp sampling, gradient limits, SAR calculation, and vendor-specific constraints. Signal, MTF, PSF, and timing values are educational simulations, not clinical recommendations.

ADVANCED 3D MODEL · COHERENT BALANCED STEADY STATE

Balance every gradient.
Keep every transverse history.

The spoiled-GRE lesson deliberately destroys transverse coherence. Here the net gradient area returns to zero every TR, so coherence survives, off-resonance phase accumulates, and the repeating vector cycle creates bright passbands separated by dark signal nulls.

SPOILED GREResidual Mxy is canceled.

The next pulse remembers mainly longitudinal recovery.

BALANCED SSFPEvery gradient has zero net area.

The next RF pulse receives a coherent 3D vector with longitudinal and transverse history.

CONSEQUENCEOff-resonance becomes periodic contrast.

Signal nulls repeat every 1/TR and move when RF phase cycling changes.

RF-PHASE-ALIGNED FRAME · TE = TR / 2 · NORMALIZED M / M₀ Closed steady-state cycle · central passband

The analytic fixed point repeats after free precession, T₁/T₂ relaxation, RF phase advance, and the next ideal RF rotation.

04 / 05 |S| 0.103 analytic repeating state

The quantitative frequency profile remains available if 3D rendering is unavailable.

DRAG TO ORBIT · + / − TO ZOOM · TAP AN OBJECT TO IDENTIFY IT

Frequency response + receiver phase EXACT NUMERIC COMPANION · SAME SOLVES AS 3D
tissue A · single phase cycle tissue B · dashed comparison four-cycle RMS when enabled tissue A receiver phase

Read magnitude and phase together: a stopband is a signal minimum with an abrupt phase transition, not a dark stripe painted at a fixed location.

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pulse 1analytic fixed point is selectedpulse 80
0 Hz −250 Hzcentral passband+250 Hz
45° signal, contrast, RF demand90°
5.0 ms 2 msband spacing 200 Hz12 ms
±250 Hz better shimlinear teaching field mapwider offset
RF PHASE INCREMENT Δφ PER TR
DISPLAYED MAGNITUDE
EXACT SINGLE-COMPARTMENT FIXED POINT · RF-ALIGNED FRAME

M+ss = Rx(α)[D(β)M+ss + c]

β = 2πΔf·TR − Δφ · E₁ = e−TR/T₁ · E₂ = e−TR/T₂ · solve (I − RxD)M = Rxc
WHITE MATTER · ECHO MAGNITUDE0.103single 180° cycle · TE 2.50 ms
receiver phase−180.0°RF-aligned convention
band spacing 1/TR200.0 Hznearest null ±100.0 Hz
distance to nearest null100.0 Hzinside a passband
post-RF vector[0.000, −0.151, 0.352]cycle closure error < 10⁻¹²
transient errorfixed pointnot assumed after one pulse
WHITE MATTER · SOLIDT₁ 850 · T₂ 80 ms|S| 0.103
CSF / FLUID · DASHEDT₁ 4000 · T₂ 2000 ms|S| 0.308
01 · BALANCEEvery gradient moment returns to zero.

Position-dependent gradient phase is rewound within each TR. Residual off-resonance phase is not.

02 · REMEMBERThe next pulse receives a full vector.

Both transverse coherence and longitudinal recovery enter the following RF rotation, so the state is three-dimensional.

03 · REPEATOne affine map reaches a fixed cycle.

The steady state is solved exactly; the optional pulse history shows why it is not established after the first excitation.

04 · FORM BANDSNulls recur every 1/TR.

RF phase cycling translates the periodic response. Separate phase-cycled acquisitions can reduce, but do not prevent, the underlying off-resonance sensitivity.

MODEL BOUNDARY · IDEAL SINGLE-COMPARTMENT bSSFP, NOT A CLINICAL SEQUENCE DESIGNER

Each displayed frequency uses one normalized isochromat with monoexponential T₁/T₂ relaxation, instantaneous nonselective RF, a constant phase increment, and perfectly zero net gradient area each TR. TE is fixed at TR/2. Vectors use an RF-phase-aligned receiver frame that distributes the programmed phase increment uniformly through each TR; the physical laboratory-frame azimuth is not plotted. The 3D frequency–I/Q manifold maps a declared linear off-resonance ramp to independent steady states: frequency is the long axis, I/Q occupy each perpendicular plane, and radius is relative echo magnitude. These are data coordinates, not anatomy, a measured B₀ map, or a reconstructed patient image. Four-cycle RMS combines four separate magnitude acquisitions and has no single coherent magnetization vector. The model excludes intravoxel frequency distributions, slice profile, B₁ error, finite RF duration, eddy-current imbalance, gradient first-moment flow effects, motion, exchange, magnetization transfer, diffusion, chemical multi-peaks, noise, coil sensitivity, k-space ordering, transient catalyzation, SAR calculation, and reconstruction. Real bSSFP names and implementations vary by vendor.

ADVANCED 3D BOLD fMRI · NEURAL INPUT → HEMODYNAMICS → T₂* SIGNAL

The scanner does not measure neurons firing.
It measures a delayed vascular consequence.

Drive one declared cortical region, follow the extended Balloon-model states, and watch changing deoxyhemoglobin alter local susceptibility, spin coherence, gradient-echo magnitude, and the sampled EPI time series. Neural input, physiology, MR contrast, acquisition, and statistical interpretation remain separate objects.

Three-dimensional BOLD neurovascular and T₂-star signal model
01 · DECLARED NEURAL INPUT A task box drives one modeled cortical region. The yellow activity cue is an input to the model. It is not a measured neuron, a BOLD value, or an activation-statistic result.
CONTINUOUS FORWARD MODEL + DISCRETE VOLUME SAMPLES t = 0.0 s · baseline · volume 0
LATEST ACQUIRED SYNTHETIC AXIAL EPI VOLUME No sampled signal change at baseline

The grayscale background is fixed synthetic anatomy. The colored patch displays the latest TR-sampled volume, while the chart cursor retains the continuous state; neither is a statistical activation map or patient image.

READ THE 3D CHAIN · The translucent brain and voxel are spatial context. Yellow pulses declare modeled neural input. Mint vessel flow and coral deoxyhemoglobin beads are hidden physiological states. Distorted field rings and spin arrows expose an MR consequence that is normally invisible. The final volume cards are discrete samples taken at TR; they are not neurons, blood cells, or proof of activation. Drag to orbit, use the visible camera controls, or tap an object for its exact meaning.

ADVANCED 3D ARTERIAL SPIN LABELING · RF LABEL → TRANSIT → DELIVERY → CBF

Turn arterial blood into a tracer.
Then subtract away almost everything else.

Run one quantitative pseudo-continuous ASL experiment from the neck to a cortical voxel. A control/label RF preparation creates the difference, arterial transit delays delivery, blood T₁ erases part of the tag, and a paired subtraction exposes a signal only a few tenths of one percent of M₀ before the consensus single-PLD equation estimates cerebral blood flow.

Three-dimensional ASL labeling, transit, subtraction, and perfusion model
01 · CONTROL / LABEL PREPARATION RF and gradient pulses mark arterial blood below the brain. The label condition inverts the flowing arterial ensemble relative to its control partner. This is magnetic preparation, not an injected substance.
ONE LABEL TRAIN · ARRIVAL WINDOW · ACQUISITION t = 0.00 s · label train begins · no delivery yet
PAIRED SYNTHETIC VOLUMES · IDENTICAL DISPLAY SCALE Control and label are almost identical before subtraction.

The control and label anatomy share one magnitude scale. Their difference uses a separately declared amplified display so the tiny modeled perfusion signal is visible; the CBF panel applies the displayed equation to that synthetic difference.

READ THE 3D CHAIN · The lower plane represents a pCASL RF-plus-gradient preparation. Moving beads are synchronized spin-packet cues: coral means label difference, fading toward gray as T₁ erases it. The vessel tree is schematic physical transport; the highlighted cortical voxel is the modeled delivery region. Control and label cards are separate acquisitions, while the difference card is a calculation. Drag to orbit, use the camera controls, or tap any object for its exact role and boundary.

ADVANCED 3D TIME-OF-FLIGHT MRA · TRANSPORT × REPEATED SPOILED GRE

Fresh blood enters bright.
RF history spends that advantage.

Follow one constant-velocity blood cohort across a hard 3D excitation slab. Physical transport decides where each RF event meets the cohort; the exact spoiled-GRE recurrence decides its remaining Mz; and a calculated maximum-intensity projection shows how that depth-dependent signal becomes a bright-blood angiographic display.

01 · TRANSPORT THROUGH THE SLAB Normal velocity sets RF encounters per transit. The rings mark where one synchronized cohort meets successive whole-slab RF pulses. They are time samples, not separate RF sheets.
Three-dimensional TOF inflow and saturation model
EXACT STROBOSCOPIC DEPTH PROFILE · DISTANCE FROM ENTRY 5.00 mm per TR · 12 RF encounters · distal blood/background 2.5×
Blood and stationary-background spoiled-GRE signal through the TOF slab The blood curve begins with fresh inflowing magnetization and changes after each RF pulse. The background curve is the local stationary spoiled-GRE steady-state signal.
flowing blood signal stationary background signal cohort position at an RF event

Why the steps are real in this model: the plot freezes a continuous plug-flow column at one RF instant. Spins that entered during the same TR share an RF count. A real voxel, slice profile, pulsatile velocity distribution, and asynchronous entry can smooth this ideal stroboscopic pattern.

READ THE 3D CAUSAL CHAIN · Each stage button replaces the central geometry so unrelated spaces cannot compete for room. The transparent box is a selected slab, not scanner hardware. A physical vessel crosses it at the chosen angle; vector length and color encode pre-RF Mz; rings locate one cohort at successive RF instants; the coral plane is an ideal 90° preparation outside the positive slab face; and Volume → MIP keeps the calculated source stack visibly separate from its max-over-Y projection. Drag to orbit, zoom, or tap any object for its exact meaning.

ADVANCED 3D PHASE-CONTRAST FLOW · BALANCED M₀, OPPOSITE M₁

Stationary phase cancels.
Velocity survives the subtraction.

Follow one steady through-plane velocity component from a transparent vessel, through two opposite bipolar encodings, into two measured I + iQ vectors and a wrapped velocity map. The physical flow, gradient moments, phase subtraction, VENC, aliasing, and integrated volume flow all share one exact state.

01 · PHYSICAL FLOW PROFILE The center of the ideal laminar profile moves fastest. Velocity is physical displacement per second along the vessel. The arrows are not gradient force, electron current, or k-space motion.
Three-dimensional phase-contrast velocity-encoding model
TWO RECTANGULAR BIPOLAR ENCODINGS · TIME IS HORIZONTALM₀,A = M₀,B = 0 · ΔM₁ +28.8 mT·ms²/m
Opposite bipolar gradient encodings and their first moments Encoding A has a negative first lobe followed by a positive lobe. Encoding B reverses both signs. Each has zero net area and opposite first moment. A · M₁ +14.4 B · M₁ −14.4 δ 1.00 ms · center separation 1.80 ms

READ THE 3D CAUSAL CHAIN · Each stage button replaces the central geometry. The tube contains the physical velocity profile. The two depth-separated moment volumes are acquisition-time diagrams—not gradients located in the vessel. The volumetric I/Q clocks show SA, SB, conjugation, and their wrapped product. The final relief surfaces use height and hue from the same signed 61 × 61 velocity arrays integrated for mL/s; violet marks wrapped samples. Drag to orbit, use visible zoom controls, or tap an object for its exact meaning.

ADVANCED 3D DIFFUSION EXPERIMENT · PULSED-GRADIENT SPIN ECHO

Stationary spins refocus. Moving water keeps a phase memory.

Orbit a magnified displacement-space model. The first gradient labels position with phase; the 180° pulse reverses that phase ordering; the matched second lobe cancels it only if a spin has not changed position along the selected direction.

06 / 06 · ECHO MEASURE THE PHASE-DISPERSED ECHO The stationary-position phase has cancelled. Displacement along the gradient leaves residual phase, so the vector sum is smaller than the b = 0 reference.
Three-dimensional diffusion encoding model

Drag to orbit · pinch, wheel, or use + / ↺ / − · tap an object to identify it · molecular displacement is magnified, not voxel scale

100%
32.6 mT/m 0both matched lobes80 mT/m
20 ms 4 msarea per lobe30 ms
40 ms 20 ms minleading-edge interval80 ms

GRADIENT WEIGHTING32.6 mT/m, δ 20 ms, and Δ 40 ms produce b = 1014 s/mm² in this ideal rectangular-lobe model.

−180°rotate in XY+180°
−90°tilt toward Z+90°

DIRECTIONAL QUESTIONg = [+1.00, +0.00, +0.00], parallel to the tensor’s fixed X principal axis.

1.50 × 10⁻³ mm²/s 0.10tensor eigenvalue3.00
0.40 × 10⁻³ mm²/s 0.10two equal transverse values3.00

AXIALLY SYMMETRIC TEACHING TENSORD∥ is 3.75× D⊥. The ellipsoid is longer along physical X; it is a voxel-scale statistical model, not a drawn axon.

RECTANGULAR-LOBE MODEL

b = (γGδ)²(Δ − δ/3)

S(b,g) / S₀ = e−bDapp(g)

γ uses radians per second per tesla inside b. Dapp = gᵀDg probes one direction through the tensor. The effective finite-lobe diffusion-time term is Δ − δ/3 = 33.3 ms.
b-value1014 s/mm²gradient timing and amplitude
apparent diffusion Dapp1.500 × 10⁻³ mm²/stensor projected onto g
Gaussian phase spread σφ1.74 rad√(2bDapp)
ideal diffusion signal S/S₀0.21878.2% attenuated before other contrast and noise
01 · FIRST LOBEPosition → phase label

G along g creates a position-dependent frequency offset. Integrating for δ writes phase proportional to g·r.

02 · DISPLACEMENTWater samples its environment

Between the lobe centers, molecules change position statistically. The 3D paths magnify micrometre-scale displacement so direction can be seen.

03 · SECOND LOBENew position → residual phase

After the 180° reversal, the matched lobe cancels stationary phase. Motion along g leaves phase proportional to the projected displacement.

04 · ECHOPhase spread → signal loss

The coil receives one vector sum. Broad residual phases cancel more, so larger bDapp lowers the ideal coherent echo.

QUANTITATIVE 3D DTI · MULTI-DIRECTION ACQUISITION → SIX-PARAMETER FIT

One direction measures one projection. A direction set estimates a tensor.

Continue directly from the pulsed-gradient experiment. Every diffusion-weighted direction supplies one value of gᵀDg. Combine non-collinear measurements, fit all six independent tensor elements, diagonalize D, and inspect where a single tensor fails.

Interactive 3D diffusion-tensor acquisition and fit
01 · DECLARED DIFFUSION SOURCE A rotated Gaussian tensor defines directional diffusion. Compare displacement spread √λ, directional ADC gᵀDg, and signal exp(−b gᵀDg). Each panel uses its own linear display scale, so compare shape and direction—not absolute size.
DIRECTION-BY-DIRECTION LEDGER Measurement 01 / 12 · g = [+0.707, +0.707, 0.000] The selected axis contributes one row to the log-linear design matrix. The opposite direction is equivalent in this ideal tensor model.
01 / 12 first axisone acquired DWIlast axis

READ THE 3D SPACES · The source and fitted ellipsoids live in one voxel’s physical coordinate frame. Gradient arrows are encoding directions, not water motion. The direction shell is a measurement design, not k-space. Drag to orbit, zoom, or tap an object for its exact role.

FORMULA EXPLAINER

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READ THE EQUATION IN WORDS

LIVE PHYSICS PICTURE

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    Symbols & units

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    Capitalization is part of the unit: M means mega (10⁶), while m can mean milli (10⁻³) or metre depending on its position.

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        MRI LANGUAGE LENS · ABBREVIATION & UNIT DICTIONARY

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        ENCODING
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        SELECTED OBJECT · 01 OF 04

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