Localize and prescribe first; then use the same quantitative sequence controls to acquire T₁, proton-density, T₂, FLAIR, DWI, or GRE contrast.
Theme 03 · Signal, contrast and motion
Run a visual exam from coil setup and localizers through slice prescription, sequence contrast, modeled k-space, and reconstruction—then inspect the physics underneath.
Choose receive hardware, acquire three-plane localizers, prescribe and reconstruct synthetic T1/PD/T2/FLAIR/DWI/GRE anatomy, then explore echo formation, steady states, perfusion, flow, angiography, and diffusion.
03 / SIGNAL, CONTRAST & MOTION
Wait for recovery.
Encode displacement.
Start with the integrated virtual exam: choose receive hardware, acquire localizers, prescribe a slice, tune T₁/PD/T₂/FLAIR/DWI/GRE controls, and reconstruct modeled k-space. TE, TR, flip angle, and RF history shape the available signal. BOLD fMRI turns a delayed vascular oxygenation consequence into T₂*-weighted time-series change; ASL magnetically labels arterial blood to estimate perfusion; TOF turns replacement of saturated blood into bright-vessel magnitude; phase contrast turns coherent motion into signed velocity; diffusion sensitization probes microscopic displacement. These are distinct mechanisms—not interchangeable maps.
BOLD fMRI is delayed and indirect: a vascular state changes susceptibility-related T₂* weighting, then EPI samples volumes at TR.
ASL couples label duration, ATT, PLD, T₁ survival, control−label difference, and model-based CBF.
TOF couples slab-normal transport to spoiled-GRE history, source magnitude, directional preparation, and MIP.
Phase contrast subtracts two complex measurements, decodes one component, and exposes VENC aliasing and area integration.
Microscopic displacement between lobes leaves a phase distribution; a wider distribution reduces the coherent echo.
Repeat directions to estimate anisotropy; one direction and one b-value are not a tensor fit or tract map.
3D ECHO FORMATION · ONE VOXEL IN THE ROTATING FRAME
A 180° pulse can reverse static phase spread.
It cannot rewind true T₂ loss.
Follow the same transverse ensemble through excitation, dephasing, a refocusing action, rephasing, the echo, and renewed dephasing. Switch mechanisms without changing the tissue or field spread.
A 90° excitation is represented as complete. Every visible spin packet begins along +X′ in a frame rotating near the carrier; the fast MHz carrier itself is removed.
The rotating-frame echo model needs WebGL. The synchronized timeline, equations, metrics, and controls remain available.
The pulse or gradient command is a cause. The phase fan converges later. Vertical screen position is a teaching state—not another scanner coordinate.
This teaching scrubber changes the instant being inspected. It does not change the prescribed TE, tissue, or field spread.
Longer TE gives both irreversible T₂ relaxation and unrefocused static offsets more time to reduce the gradient echo. The ideal spin echo removes the static-offset term only at its echo center.
A wider static frequency distribution fans phase faster. The 180° pulse reverses its ordering; a gradient-polarity reversal does not.
SSE(TE) / S₀ = e−TE/T₂
The 180° pulse removes the static-offset phase factor at TE; it does not restore the T₂ envelope.1/T₂* = 1/T₂ + 1/T₂′ · T₂′ = 1/(πΔfFWHM)
1/T₂* = 1/80 + 1/79.6 ms → T₂* = 39.9 msWHY THE TWO ECHOES DIFFER ·At TE 80 ms, intrinsic T₂ leaves 36.8%. A 4.0 Hz static spread reduces the gradient echo to 13.5%, while an ideal 180° pulse removes that static factor at the spin-echo center.
Static frequency offsets keep their sign.
Fast packets move ahead and slow packets fall behind. An ideal 180° pulse swaps that phase ordering, so continued evolution brings them together at TE.
True T₂ coherence keeps decaying.
The visible packet arrows shorten as the modeled coherent contribution survives. The refocusing pulse changes future phase evolution; it does not add lost coherence or reset time.
Opposite gradient area is not a 180° pulse.
A gradient echo cancels the deliberate gradient moment at TE but leaves static B₀ offsets accumulating, so its ideal envelope follows T₂*.
Choose receive hardware, localize, prescribe a slice, then acquire it.
Review the 3D RF + Gz slice-selection microscope →LIVE SEQUENCE · T₁ SPIN ECHOTR 500 · TE 15 ms
HEAD TEACHING STARTBrain, ventricles, and posterior fossa. Acquire the synthetic localizers, prescribe, then run the virtual scan.
- TRANSMIT
- built-in body transmit
- RECEIVE
- circumferential head and upper-neck receive array
- CENTER SENSITIVITY
- 1.00× relative
- UNIFORMITY PROXY
- 0%
- MEAN RECEIVE PROXY
- 0.00× effective
- PARALLEL IMAGING
- not simulated
Receive proxy = mean analytic sensitivity over one central axial elliptical mask. Nominal channel count is a hardware label, not a multiplier. A posterior partner changes the modeled sensitivity term. This does not predict clinical SNR or usable acceleration.
Acquire the survey first. Localizers reveal internal synthetic anatomy relative to scanner coordinates; the external landmark alone does not prescribe a slice.
ONE 3D MODEL, THREE REPARAMETERISATIONSAll three surveys and every prescribed slice sample the same code-native 3D tissue field in patient millimetres: R/L positive toward L, A/P positive toward P, H/F positive toward F, measured from each region's modelled isocentre. Axial reads (R/L, A/P) at a fixed H/F, sagittal reads (A/P, H/F) at a fixed R/L, coronal reads (R/L, H/F) at a fixed A/P. R→L runs left to right; axial A→P and sagittal/coronal H→F run top to bottom. Positive slice position points toward L, P, or F for sagittal, coronal, or axial prescriptions, and its limit is that region's own half-extent along the selected normal. Each survey is a soft-maximum (power-mean, p = 3) projection of tissue proton density ρ through a 15 mm slab. Every displayed scout voxel uses 2 × 2 in-plane midpoint samples and seven through-slab samples—28 samples total. It is a declared projection, not a simulated pulse sequence, so it carries no TR, TE, TI, flip angle, relaxation, diffusion, coil weighting, or noise. Survey brightness uses a declared display window (ρ 0.56–1.02, γ 0.90); the prescription overlay is drawn in true millimetres at each survey's own scale.
AT THE TEACHING STARTChange slice thickness, FOV, matrix, or NEX. This readout separates resolution, SNR, coverage, and time so “brighter” is not mistaken for “better.”
- CYCLE
- —
- LINES / WINDOW
- all lines
- RESIDUAL MOTION
- 0.0 mm
- WALL-CLOCK PROXY
- 1:04
NO PERIODIC MOTIONThe k-space rows remain mutually consistent. Choose Cardiac or Respiratory, then compare gating on and off in the reconstructed image.
CONTROLS CHANGEDThis result preserves the acquired state. Run the scan again to apply the new sequence or prescription.
WHAT IS QUANTITATIVETR, TE, TI, flip angle, b/ADC signal equations; FOV/matrix pixel size; voxel volume; slice-stack coverage; one-line-per-TR base time; SNR scaling with voxel volume and √NEX while other factors are held fixed; the exact Fourier translation phase applied to each modeled kᵧ line; the Fourier transform of the displayed center-slice synthetic source; and the plane geometry itself — every survey, overlay, embedded 3D slice, and acquired image is the same 3D field sampled through one millimetre coordinate map.
WHAT IS ILLUSTRATIVEThe ten study buttons are starting states, not protocols. Analytic coil sensitivity, deterministic complex k-space noise, the cardiac/respiratory waveform, trigger acceptance and wall-clock proxy, whole-slice rigid displacement, procedural anatomy, derived display shells, localizer projection/window, partial-volume quadrature, preview resolution, per-region scene scale, nominal channel labels, and compressed playback are teaching models. Slice thickness can improve this model's SNR and increase through-plane averaging; fixed display windowing means it does not guarantee a brighter image.
WHAT IS OMITTEDThe other prescribed slices and repeated NEX lines are not separately reconstructed. The anatomy is hand-declared—not segmented, measured, atlas-derived, registered, diagnostic, or population-normal. Motion is one rigid phase-axis translation, not organ deformation, flow, temporal cine reconstruction, navigator design, arrhythmia classification, prospective controller behavior, or a patient-specific quiet window. Also omitted: measured B₀/B₁/coil maps, channel noise covariance, bandwidth changes, parallel reconstruction, safety screening/limits, calibration, and vendor protocol logic. Nothing here recommends a clinical protocol.
S = ρH(1 − e−TR/T₁)e−TE/T₂
The 180° pulse refocuses static dephasing, so ideal echo amplitude follows T₂ rather than T₂*.Short TR samples tissues before full longitudinal recovery, strengthening T₁ differences. Short TE limits T₂ decay, so the current spin echo is predominantly T₁ weighted.
MODEL BOUNDARY · SYNTHETIC ANATOMY, NOT A PATIENT OR PROTOCOL RECOMMENDATION. Every pixel belongs to one ideal nonexchanging tissue compartment with illustrative values near 3 T. Spin echo uses ρ(1−e−TR/T₁)e−TE/T₂; FLAIR adds one ideal inversion-prepared repeated cycle; DWI multiplies the same spin-echo baseline by e−b·ADC; GRE uses the spoiled steady state and T₂*. The grayscale uses one fixed relative-signal display mapping, so numeric comparisons remain available without per-preset auto-windowing. Real anatomy and contrast also depend on field strength, sequence family, echo trains and k-space ordering, inversion and fat-suppression profiles, diffusion direction, T₂ shine-through, perfusion, flow, exchange, coils, noise, artifacts, reconstruction, pathology, safety limits, and vendor timing constraints.
ADVANCED 3D MODEL · T₁ SATURATION ACROSS REPETITIONS
One RF pulse is simple.
The history creates the steady state.
Follow one ideally spoiled gradient-echo experiment pulse by pulse. The vector shortens inside a reference sphere, the history plot records the exact recurrence, and the Ernst protractor shows which flip angle maximizes ideal steady-state signal for the selected tissue and TR.
The experiment begins at equilibrium: Mz⁻ = 1 before the first RF pulse. The translucent sphere is only the M₀ reference boundary.
The quantitative history plot remains available if 3D rendering is unavailable.
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Important: these curves show magnetization state, not TE-weighted signal. TE scales the echo readout after each pulse; it does not change convergence toward the spoiled-GRE steady state.
Mn+1− = 1 − (1 − Mn− cosα)E₁
E₁ = e−TR/T₁ · fixed point Mss− = (1 − E₁)/(1 − E₁ cosα) · αE = cos−1(E₁)The first repetition starts at equilibrium. Later pulses begin from the recovery left by every earlier pulse.
Instantaneous ideal RF creates Mxy = Mz⁻ sinα and leaves Mz⁺ = Mz⁻ cosα.
T₂* reduces the measured echo. The recurrence then applies coherent Mxy → 0 before the next pulse without claiming a gradient- or RF-spoiling mechanism.
During TR, Mz returns toward M₀. Repeating the same map approaches one fixed point at rate E₁ cosα.
One normalized isochromat per tissue is shown in the rotating frame, with instantaneous nonselective RF, perfect loss of coherent transverse magnetization between repetitions, monoexponential T₁ recovery, and magnitude-only T₂* echo weighting. T₁ recovery during the short TE is folded into the repetition map. The model excludes coherent/balanced SSFP, RF phase cycling details, B₁ error, slice profile, inflow and flow, magnetization transfer, diffusion, exchange, multiple compartments, off-resonance banding, imaging gradients, noise, and reconstruction. Z/B₀ is the spin-frame reference here, not a patient-orientation claim.
ADVANCED 3D MODEL · INVERSION RECOVERY FAMILY
Invert first.
Wait for one tissue to disappear.
An inversion preparation sends longitudinal magnetization below zero. Tissues recover at different T₁ rates, so a readout placed at the right inversion time can null fat, null fluid, or preserve the sign that ordinary magnitude display folds away.
Efficiency controls the effective longitudinal inversion; it is not drawn as a fictitious hard-pulse angle.
The finite-TR fixed cycle shifts that crossing from the familiar long-TR T₁ ln 2 result.
A phase-sensitive display preserves negative-versus-positive recovery; magnitude folds both sides above zero.
The five declared materials rise through the shared zero plane at different times; fat is at its finite-TR crossing near TI 243 ms.
The signed TI-response plot remains available if 3D rendering is unavailable.
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Upper plot: signed Mz immediately before the readout. Lower plot: the chosen signed or magnitude echo after flip-angle, proton-density, and TE weighting.
Mz(TI) = 1 − cinvETR − (1 − cinv)ETI1 − cinvcos α ETR
cinv = 1 − 2η · ETI = e−TI/T₁ · S = ρ Mz(TI) sin α e−TE/T₂(or T₂*)η = 1 maps +Mz to −Mz. Lower η reduces that effective longitudinal displacement and changes the null.
Fat crosses zero before white matter, gray matter, and CSF in this declared 3 T teaching table.
The readout flip converts Mz into Mxy. Spin echo uses T₂; gradient echo uses T₂* in this model.
Signed mode retains polarity; magnitude mode applies an absolute value after the same physical signal calculation.
Every material is one normalized, nonexchanging compartment with monoexponential T₁, T₂, and T₂* constants. Inversion and readout are instantaneous and spatially uniform. η is an effective longitudinal action, not a literal pulse angle or a model of adiabatic-pulse dynamics. The repeated mode contains one readout per TR with exact fixed-cycle history; fully recovered mode forces Mpre = M₀. The colored lanes and tiles are declared material samples, not anatomy or a diagnostic image. “STIR” and “FLAIR” presets isolate their nulling mechanism; they are not scanner protocols. “Signed / PSIR-like” preserves ideal signal polarity but does not model a reference acquisition, phase errors, or PSIR reconstruction. Excluded effects include slice profile, B₁ variation, finite pulse duration, magnetization transfer, exchange, multi-component fat, CSF flow, FSE echo trains, stimulated echoes, k-space ordering, coils, noise, motion, SAR, parallel imaging, and vendor timing constraints.
ADVANCED 3D MODEL · FSE / TSE / RARE · EXTENDED PHASE GRAPH
One excitation.
Many echoes, many pathways.
A fast spin-echo train is not one spin echo copied many times. Every refocusing pulse mixes transverse and longitudinal configuration states. Those coherent and stimulated-echo pathways determine each echo; assigning different echoes to phase-encode lines then changes contrast, point spread, and scan time.
EPG order k records accumulated gradient phase area. It is dimensionless pathway bookkeeping—not an imaging ky coordinate.
Sub-180° pulses store part of the history longitudinally and recall it later as stimulated echoes.
The resulting complex echo weighting is an MTF; its inverse transform is the phase-direction PSF.
EPG CONFIGURATION ORDER k tracks dephasing pathways inside one modeled compartment. IMAGING ky is a commanded spatial-frequency address acquired on one echo. The dashed bridge is a data-flow link, not an equality.
The receiver samples only F₀ at 80 ms. Other transverse and longitudinal orders preserve hidden history for later echoes.
The exact echo-train, MTF, and PSF plots remain available if 3D rendering is unavailable.
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Ωkafter RF = T(α,φ)Ωkbefore RF · Fk+ → Fk+1+
Sn = ρF0(n·ESP) · W(ky) = Se(ky)/maxe|Se| · PSF = DFT−1{W}Relaxation scales F states by E₂ and Z states by E₁; recovery adds only to equilibrium Z₀.
A 180° CPMG pulse swaps transverse pathways. Lower angles also create Z storage and later stimulated echoes.
The receiver sees the signed complex sum at order zero, not the hidden higher-order states individually.
Center ky largely sets effective contrast; variation over ky determines the PSF, blur, ringing, and possible phase modulation.
The EPG engine is on-resonance, nonselective, single-compartment, and monoexponential. RF pulses are instantaneous and spatially uniform; every half-echo interval receives one identical positive unit gradient-order shift. CPMG and CP specify ideal RF-axis relationships. The “declared ramp” is a transparent exponential 180°-to-floor teaching schedule—not a vendor optimizer, prescribed-signal algorithm, or claim of an executable protocol. The imaging panel separately maps the solved echo train into declared grouped one-dimensional phase-encode bands across 128 lines. It is not the EPG configuration axis, a universal view order, or a two-dimensional anatomy reconstruction. TR is used only for scan-time accounting because every displayed train begins at equilibrium; inter-train saturation is excluded. Also excluded are slice profile and selective-pulse crushers, B₁/B₀ distributions, finite RF duration, chemical shift, multiple compartments, exchange, magnetization transfer, diffusion, flow, motion, noise, coils, parallel imaging, partial Fourier, echo sharing, ramp sampling, gradient limits, SAR calculation, and vendor-specific constraints. Signal, MTF, PSF, and timing values are educational simulations, not clinical recommendations.
ADVANCED 3D MODEL · COHERENT BALANCED STEADY STATE
Balance every gradient.
Keep every transverse history.
The spoiled-GRE lesson deliberately destroys transverse coherence. Here the net gradient area returns to zero every TR, so coherence survives, off-resonance phase accumulates, and the repeating vector cycle creates bright passbands separated by dark signal nulls.
The next pulse remembers mainly longitudinal recovery.
The next RF pulse receives a coherent 3D vector with longitudinal and transverse history.
Signal nulls repeat every 1/TR and move when RF phase cycling changes.
The analytic fixed point repeats after free precession, T₁/T₂ relaxation, RF phase advance, and the next ideal RF rotation.
The quantitative frequency profile remains available if 3D rendering is unavailable.
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Read magnitude and phase together: a stopband is a signal minimum with an abrupt phase transition, not a dark stripe painted at a fixed location.
M+ss = Rx(α)[D(β)M+ss + c]
β = 2πΔf·TR − Δφ · E₁ = e−TR/T₁ · E₂ = e−TR/T₂ · solve (I − RxD)M = RxcPosition-dependent gradient phase is rewound within each TR. Residual off-resonance phase is not.
Both transverse coherence and longitudinal recovery enter the following RF rotation, so the state is three-dimensional.
The steady state is solved exactly; the optional pulse history shows why it is not established after the first excitation.
RF phase cycling translates the periodic response. Separate phase-cycled acquisitions can reduce, but do not prevent, the underlying off-resonance sensitivity.
Each displayed frequency uses one normalized isochromat with monoexponential T₁/T₂ relaxation, instantaneous nonselective RF, a constant phase increment, and perfectly zero net gradient area each TR. TE is fixed at TR/2. Vectors use an RF-phase-aligned receiver frame that distributes the programmed phase increment uniformly through each TR; the physical laboratory-frame azimuth is not plotted. The 3D frequency–I/Q manifold maps a declared linear off-resonance ramp to independent steady states: frequency is the long axis, I/Q occupy each perpendicular plane, and radius is relative echo magnitude. These are data coordinates, not anatomy, a measured B₀ map, or a reconstructed patient image. Four-cycle RMS combines four separate magnitude acquisitions and has no single coherent magnetization vector. The model excludes intravoxel frequency distributions, slice profile, B₁ error, finite RF duration, eddy-current imbalance, gradient first-moment flow effects, motion, exchange, magnetization transfer, diffusion, chemical multi-peaks, noise, coil sensitivity, k-space ordering, transient catalyzation, SAR calculation, and reconstruction. Real bSSFP names and implementations vary by vendor.
ADVANCED 3D BOLD fMRI · NEURAL INPUT → HEMODYNAMICS → T₂* SIGNAL
The scanner does not measure neurons firing.
It measures a delayed vascular consequence.
Drive one declared cortical region, follow the extended Balloon-model states, and watch changing deoxyhemoglobin alter local susceptibility, spin coherence, gradient-echo magnitude, and the sampled EPI time series. Neural input, physiology, MR contrast, acquisition, and statistical interpretation remain separate objects.
The grayscale background is fixed synthetic anatomy. The colored patch displays the latest TR-sampled volume, while the chart cursor retains the continuous state; neither is a statistical activation map or patient image.
READ THE 3D CHAIN · The translucent brain and voxel are spatial context. Yellow pulses declare modeled neural input. Mint vessel flow and coral deoxyhemoglobin beads are hidden physiological states. Distorted field rings and spin arrows expose an MR consequence that is normally invisible. The final volume cards are discrete samples taken at TR; they are not neurons, blood cells, or proof of activation. Drag to orbit, use the visible camera controls, or tap an object for its exact meaning.
ADVANCED 3D ARTERIAL SPIN LABELING · RF LABEL → TRANSIT → DELIVERY → CBF
Turn arterial blood into a tracer.
Then subtract away almost everything else.
Run one quantitative pseudo-continuous ASL experiment from the neck to a cortical voxel. A control/label RF preparation creates the difference, arterial transit delays delivery, blood T₁ erases part of the tag, and a paired subtraction exposes a signal only a few tenths of one percent of M₀ before the consensus single-PLD equation estimates cerebral blood flow.
The control and label anatomy share one magnitude scale. Their difference uses a separately declared amplified display so the tiny modeled perfusion signal is visible; the CBF panel applies the displayed equation to that synthetic difference.
READ THE 3D CHAIN · The lower plane represents a pCASL RF-plus-gradient preparation. Moving beads are synchronized spin-packet cues: coral means label difference, fading toward gray as T₁ erases it. The vessel tree is schematic physical transport; the highlighted cortical voxel is the modeled delivery region. Control and label cards are separate acquisitions, while the difference card is a calculation. Drag to orbit, use the camera controls, or tap any object for its exact role and boundary.
The scanner is preparing flowing arterial magnetization below the brain.
At t = 0.00 s the label window has opened. The first prepared packet reaches the modeled voxel after the selected arterial transit time.
DELIVERY RATEWith timing and efficiency fixed, ΔM is linear in the declared flow. This does not make image brightness a direct flow unit until the model, M₀ reference, and constants are applied.
THREE DIFFERENT CLOCKSτ creates the bolus. ATT says when its front reaches tissue. PLD waits from the end of labeling to acquisition. Full delivery in this model requires PLD ≥ ATT.
PREPARATION × RELAXATIONα scales how much difference is created. Blood T₁ controls how fast that difference decays while packets travel and wait; neither control changes the declared true flow.
a(t) = clamp[t − ATT, 0, τ]
ΔM(t)/M₀ = 2α(F/6000)T₁b/λ [e−(t−a)/T₁b − e−t/T₁b]
CBF = 6000λΔM ePLD/T₁b2αT₁bM₀(1 − e−τ/T₁b)
At acquisition: t = τ + PLD = 3.60 s · full bolus delivered · calculate ΔM and invertThe label/control preparation changes magnetization, not chemistry, speed, oxygenation, or blood-flow physiology.
Waiting long enough improves delivery completeness, but a longer delay also leaves less label difference through relaxation.
Motion and physiological changes between real control/label images would not cancel perfectly; this ideal pair is noiseless and registered.
The single-PLD inversion is exact only inside the displayed retained-label assumptions. Too-early acquisition creates an explicit negative bias here.
This is a deterministic single-region pCASL teaching experiment, not patient data or protocol advice. It represents continuous label production from 0 to τ, one fixed arterial transit time, exponential blood-label decay, complete retention after delivery (equivalent here to T₁app = T₁b with outflow neglected), M₀ = 1, λ = 0.9 mL/g, and the displayed control−label sign. When PLD ≥ ATT, its acquisition expression reduces exactly to the 2015 consensus single-PLD equation; when PLD < ATT, it deliberately exposes incomplete-bolus underestimation. The RF rings and gradient arrows are a qualitative pCASL preparation cue—not a Bloch solution or pulse train design. It omits dispersion, multiple arrival paths, tissue/blood exchange kinetics, tissue T₁, capillary permeability, venous outflow, macrovascular signal, background suppression, vascular crushing, B₀/B₁ error, magnetization transfer asymmetry, motion, cardiac and respiratory variation, noise, receive sensitivity, partial volume, labeling-plane geometry, readout timing, k-space, reconstruction, calibration uncertainty, pathology inference, and multi-PLD fitting. The uniformly colored CBF glyph is one synthetic modeled region—not estimated territories or diagnostic anatomy.
ADVANCED 3D TIME-OF-FLIGHT MRA · TRANSPORT × REPEATED SPOILED GRE
Fresh blood enters bright.
RF history spends that advantage.
Follow one constant-velocity blood cohort across a hard 3D excitation slab. Physical transport decides where each RF event meets the cohort; the exact spoiled-GRE recurrence decides its remaining Mz; and a calculated maximum-intensity projection shows how that depth-dependent signal becomes a bright-blood angiographic display.
Why the steps are real in this model: the plot freezes a continuous plug-flow column at one RF instant. Spins that entered during the same TR share an RF count. A real voxel, slice profile, pulsatile velocity distribution, and asynchronous entry can smooth this ideal stroboscopic pattern.
READ THE 3D CAUSAL CHAIN · Each stage button replaces the central geometry so unrelated spaces cannot compete for room. The transparent box is a selected slab, not scanner hardware. A physical vessel crosses it at the chosen angle; vector length and color encode pre-RF Mz; rings locate one cohort at successive RF instants; the coral plane is an ideal 90° preparation outside the positive slab face; and Volume → MIP keeps the calculated source stack visibly separate from its max-over-Y projection. Drag to orbit, zoom, or tap any object for its exact meaning.
Blood crosses the slab before reaching the stationary fixed point.
At 25 cm/s normal flow, one cohort advances 5.00 mm per TR and meets 12 RF pulses inside the 60 mm slab. Its distal signal remains above stationary gray matter.
TRANSPORT CLOCKv⊥ = 25.0 cm/s · distance/TR = 5.00 mm · transit = 240 ms · 12 RF encounters.
SPOILED-GRE SIGNAL HISTORYEach RF tip uses the local α, perfect spoiling removes net transverse history, T₁ recovers during TR, and TE adds magnitude-only T₂* weighting.
v⊥ = |v|cosθ · Δs = v⊥TR · N ≈ ⌈L/Δs⌉
Mz,n+1− = 1 − [1 − Mz,n−cosαn]e−TR/T₁
Sn = ρMz,n−sinαne−TE/T₂* · MIP(x,z) = maxy|S(x,y,z)|
25 cm/s × 20 ms = 5.00 mm/TR · 60 mm / 5.00 = 12 RF encountersSpeed changes how many RF histories fit inside the slab. Signal still depends on flip angle, TR, T₁, TE/T₂*, proton density, slab thickness, direction, and preparation history.
Only v⊥ replaces blood through these slab faces. At θ = 90° this boundary model supplies no fresh face entry, so resident blood approaches its spoiled-GRE fixed point even if total speed is large.
The teaching ramp rises linearly from 70% of nominal at −Z to 130% at +Z. It can compensate one preferred direction; reversing flow makes blood meet the high-flip end first.
The final plane keeps the greatest source magnitude along +Y for every X/Z ray. It can emphasize bright vessels but discards depth and can also retain any bright background or contaminant.
This is a noiseless stroboscopic constant-velocity plug-flow model with a straight vessel crossing a rectangular spoiled-GRE slab. RF is instantaneous, the slice profile is hard, transverse coherence is perfectly spoiled before every repetition, T₁ recovery is monoexponential, and TE contributes only magnitude T₂* decay. Flow angle changes only the slab-normal component |v|cosθ; lateral FOV entry is not modeled. At exactly 90° the resident compartment is placed at its local steady state. The optional TONE profile is a declared linear teaching ramp from 70% to 130% of nominal flip, not a vendor pulse design. The coral preparation is an ideal 90° band that sets Mz = 0; blood then recovers across a fixed 8 mm gap. The final volume uses an analytic synthetic vessel mask and the displayed MIP is a direct maximum of its calculated magnitude array—not acquired k-space, a ray-traced anatomical scan, a patient image, or a stenosis measurement. Excluded effects include pulsatility, acceleration, parabolic/sheared/turbulent flow, intravoxel dephasing, gradient moment and flow compensation, finite RF profiles, B₀/B₁ error, magnetization transfer, exchange, contrast agent, cardiac gating, MOTSA/multislab junctions, partial volume, noise, parallel imaging, reconstruction filters, pathology, and clinical protocol validation.
ADVANCED 3D PHASE-CONTRAST FLOW · BALANCED M₀, OPPOSITE M₁
Stationary phase cancels.
Velocity survives the subtraction.
Follow one steady through-plane velocity component from a transparent vessel, through two opposite bipolar encodings, into two measured I + iQ vectors and a wrapped velocity map. The physical flow, gradient moments, phase subtraction, VENC, aliasing, and integrated volume flow all share one exact state.
READ THE 3D CAUSAL CHAIN · Each stage button replaces the central geometry. The tube contains the physical velocity profile. The two depth-separated moment volumes are acquisition-time diagrams—not gradients located in the vessel. The volumetric I/Q clocks show SA, SB, conjugation, and their wrapped product. The final relief surfaces use height and hue from the same signed 61 × 61 velocity arrays integrated for mL/s; violet marks wrapped samples. Drag to orbit, use visible zoom controls, or tap an object for its exact meaning.
The measured center velocity keeps its correct sign.
A +28.0 cm/s center velocity creates a wrapped phase difference below +π. The recovered map remains +28.0 cm/s at the center and its lumen integral remains consistent with the ideal profile.
Velocity changes displacement during both encodings. It does not alter the gradient waveform, VENC, vessel diameter, or common background phase.
VELOCITY SENSITIVITYEach encoding has zero net area. Increasing |G|, δ, or Δ increases |M₁|, lowers VENC, and makes a given velocity rotate farther.
Diameter changes cross-sectional area and therefore mL/s at the same velocity. It does not change phase per cm/s or VENC.
Δφ = arg(SASB*) = wrap(γΔM1v)
VENC = π / (γ|ΔM1|) · vmeas = Δφ / (γΔM1)
γ × 28.8 mT·ms²/m × +28 cm/s = +123.6° wrapped phaseEqual opposite gradient area refocuses a stationary position. Because the lobes occur at different times, a spin moving at constant velocity samples different positions and retains phase proportional to the first moment.
Both measurements contain the same declared background phase. Multiplying SA by SB* subtracts their angles while their opposite velocity sensitivities add.
At speeds beyond VENC the complex angle crosses +π and reappears near −π. The displayed velocity can therefore acquire the wrong sign; it does not stick at the slider maximum.
Phase gives the encoded velocity component. Volume flow also needs a lumen mask and physical pixel area, then integrates velocity across that area. Diameter can change mL/s without changing cm/s.
This is one steady velocity component normal to an ideal circular slice. It uses two noiseless unit-magnitude complex measurements, a fixed shared background phase, equal rectangular bipolar lobes, M₀ = 0, symmetric ±M₁ encoding, constant velocity during the gradients, and either a plug or fully developed parabolic profile. The final map is an analytical synthetic phase map, not a k-space reconstruction or patient image. It excludes acceleration, pulsatility, turbulence, intravoxel phase dispersion, magnitude loss, eddy-current and concomitant-gradient phase, gradient nonlinearity, background-phase correction, partial volume, finite slice profile, noise, velocity-to-noise tradeoffs, cardiac gating, segmentation error, oblique velocity projection, three-direction encoding, pressure, wall shear stress, and 4D-flow reconstruction. A clinical VENC choice must balance alias avoidance against velocity-to-noise performance; this model is not a protocol recommendation.
ADVANCED 3D DIFFUSION EXPERIMENT · PULSED-GRADIENT SPIN ECHO
Stationary spins refocus. Moving water keeps a phase memory.
Orbit a magnified displacement-space model. The first gradient labels position with phase; the 180° pulse reverses that phase ordering; the matched second lobe cancels it only if a spin has not changed position along the selected direction.
Drag to orbit · pinch, wheel, or use + / ↺ / − · tap an object to identify it · molecular displacement is magnified, not voxel scale
GRADIENT WEIGHTING32.6 mT/m, δ 20 ms, and Δ 40 ms produce b = 1014 s/mm² in this ideal rectangular-lobe model.
DIRECTIONAL QUESTIONg = [+1.00, +0.00, +0.00], parallel to the tensor’s fixed X principal axis.
AXIALLY SYMMETRIC TEACHING TENSORD∥ is 3.75× D⊥. The ellipsoid is longer along physical X; it is a voxel-scale statistical model, not a drawn axon.
b = (γGδ)²(Δ − δ/3)
S(b,g) / S₀ = e−bDapp(g)
γ uses radians per second per tesla inside b. Dapp = gᵀDg probes one direction through the tensor. The effective finite-lobe diffusion-time term is Δ − δ/3 = 33.3 ms.G along g creates a position-dependent frequency offset. Integrating for δ writes phase proportional to g·r.
Between the lobe centers, molecules change position statistically. The 3D paths magnify micrometre-scale displacement so direction can be seen.
After the 180° reversal, the matched lobe cancels stationary phase. Motion along g leaves phase proportional to the projected displacement.
The coil receives one vector sum. Broad residual phases cancel more, so larger bDapp lowers the ideal coherent echo.
This is one ideal pulsed-gradient spin-echo experiment with two rectangular, perfectly matched lobes; a perfect 180° pulse; stationary background; Gaussian monoexponential diffusion; one axially symmetric tensor whose principal axis is fixed to physical X; no imaging gradients, eddy currents, concomitant fields, gradient nonlinearity, cross-terms, perfusion, flow, restriction-time dependence, exchange, kurtosis, noise, EPI distortion, or T₂ weighting. The paths are deterministic teaching samples in magnified displacement space—not literal tracked molecules, an axon bundle, a voxel-to-scale rendering, a diffusion-weighted clinical image, a tensor fit, or tractography. Visible ellipsoid radii follow √D to match displacement spread; they are not the eigenvalue-scaled glyph convention used by every DTI display. The per-spin arrows use the narrow-pulse phase label q·Δr, while the analytical b-value and path variance retain the rectangular finite-lobe Δ − δ/3 correction. Real DTI requires multiple non-collinear directions plus reference data and validated fitting.
QUANTITATIVE 3D DTI · MULTI-DIRECTION ACQUISITION → SIX-PARAMETER FIT
One direction measures one projection. A direction set estimates a tensor.
Continue directly from the pulsed-gradient experiment. Every diffusion-weighted direction supplies one value of gᵀDg. Combine non-collinear measurements, fit all six independent tensor elements, diagonalize D, and inspect where a single tensor fails.
READ THE 3D SPACES · The source and fitted ellipsoids live in one voxel’s physical coordinate frame. Gradient arrows are encoding directions, not water motion. The direction shell is a measurement design, not k-space. Drag to orbit, zoom, or tap an object for its exact role.
Twelve non-collinear measurements recover one rotated tensor.
Noise perturbs the log-linear fit, but the source obeys the same monoexponential model used by the estimator.
MEASUREMENT MODELEach signal is S/S₀ = exp(−b gᵀDg). Deterministic complex Gaussian noise is added before magnitude formation.
PHYSICAL ORIENTATIONThe declared symmetry axis is [+0.770, +0.539, +0.342]. Rotating it changes off-diagonal tensor elements and the directional signal pattern; it is a unique principal line only when its eigenvalue is distinctly largest.
DECLARED EIGENVALUESThe source FA is 0.762 and MD is 0.800 × 10⁻³ mm²/s before sampling and fitting.
CROSSING COMPARTMENTSThese controls are dormant for a single source. In crossing mode, two nonexchanging tensors contribute a magnitude-weighted signal mixture that one tensor cannot exactly represent at finite b.
−ln(Si/S₀)/bi = giᵀDgi = gx²Dxx + gy²Dyy + gz²Dzz + 2gxgyDxy + 2gxgzDxz + 2gygzDyz
D = V diag(λ₁, λ₂, λ₃)Vᵀ · MD = (λ₁ + λ₂ + λ₃)/3
FA = √[(3/2) Σ(λj − MD)² / Σλj²]
12 axes · b 1000 s/mm² · fitted λ = 1.700, 0.350, 0.350 × 10⁻³ mm²/sA symmetric 3 × 3 tensor has six independent elements. Six independent equations can interpolate six measurements exactly—even six noisy ones—so extra directions expose residuals and support more stable estimation.
The scalar gᵀDg is quadratic in direction, so reversing every gradient component leaves this ideal tensor signal unchanged. Real sequences can contain direction-dependent artifacts outside this model.
The orientation color uses absolute fitted principal-vector components weighted by FA. It summarizes one voxel-scale tensor estimate; it does not prove an axon, its polarity, or a connected pathway.
A finite-b sum of two exponentials is not generally one exponential. The fitted principal direction can fall between the actual component axes, and FA can decrease even though both source tensors are anisotropic.
This is a single-voxel teaching estimator. It uses one exact normalized S₀, one common scalar b-value, nested 6, 12, or 30 declared non-collinear axes, Gaussian monoexponential tensors, and deterministic independent complex Gaussian noise before magnitude formation. The fit is ordinary unweighted log-linear least squares for six symmetric tensor elements, with no positivity constraint, outlier rejection, denoising, bias correction, motion/eddy-current/susceptibility correction, gradient-nonlinearity correction, b-matrix rotation, spatial regularization, or uncertainty interval. The crossing mode is a magnitude-weighted sum of two nonexchanging Gaussian compartments and is intentionally fit with the wrong single-tensor model. Ellipsoids and radial surfaces are statistical glyphs, not axons. A principal eigenvector is an unoriented line and is reported only when λ₁ separates sufficiently from λ₂ under the declared 0.02 linearity threshold. The crossing reference is a weighted low-b moment tensor, not a finite-b ground truth. FA and MD are model-derived summaries, not tissue labels or diagnoses. Tractography, if performed clinically or in research, adds spatial propagation, stopping, uncertainty, acquisition, and model choices that this voxel experiment does not implement.